Joëlle Magné
23
Documents
Publications
Publications
Suppression of non-canonical autophagy induces endothelial dysfunction and a decrease in endothelial hemoglobin subunit alphaGordon research seminar Red blood cells- Exploring the Mechanisms Controlling Erythropoiesis and Red Blood Cell Biology in Health and Disease, Gordon research conference, Jun 2025, Newport (RI), United States
Communication dans un congrès
hal-05269796
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Non-canonical functions of autophagy proteins in cardiovascular diseases91st EAS congress, European Atherosclerosis Society, May 2023, Manheim, Germany. pp.S6, ⟨10.1016/j.atherosclerosis.2023.06.899⟩
Communication dans un congrès
hal-05269749
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Repression of MAP1LC3A during atherosclerosis progression plays an important role in the regulation of vascular smooth muscle cell phenotype86th European Atherosclerosis society congress, European Atherosclerosis Society, Jun 2020, Leiden (Netherlands), Netherlands. pp.e22-e23, ⟨10.1016/j.atherosclerosis.2020.10.080⟩
Communication dans un congrès
hal-05269833
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ATG16L1 Expression in Carotid Atherosclerotic Plaques Is Associated With Plaque Vulnerability.Gordon research conference: The Behaviour and Function of Phagocytes in Health and Disease, Gordon research conference, May 2015, Waterville Valley, United States
Communication dans un congrès
hal-05269853
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LAP AND LANDO: Non-canonical functions of autophagy proteins in cardiovascular disease2nd Immuno-Metabolic Mechanisms of Atherosclerosis Conference, Mar 2020, Nassau, Bahamas
Poster de conférence
hal-05269804
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Repression of MAP1LC3A during atherosclerosis progression plays an important role in the regulation of vascular smooth muscle cell phenotype.Vascular Discovery: From Genes to Medicine Scientific Sessions 2018, May 2018, San Francisco CA, United States
Poster de conférence
hal-05269810
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ATG16L1 Expression in Carotid Atherosclerotic Plaques Is Associated With Plaque VulnerabilityEMBO conference: Autophagy signaling and progression in health and disease, Sep 2015, Chia Laguna, Sardinia, Italy
Poster de conférence
hal-05269836
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