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ISG15 counteracts Listeria monocytogenes infection.
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Les multiples stratégies de Listeria
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Human BAHD1 promotes heterochromatic gene silencing.
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Listeria monocytogenes: a multifaceted model
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WASP-related proteins, Abi1 and Ena/VASP are required for Listeria invasion induced by the Met receptor
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Identification of substrates of the Listeria monocytogenes sortases A and B by a non-gel proteomic analysis
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Analysis of the Listeria cell wall proteome by two-dimensional nanoliquid chromatography coupled to mass spectrometry
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Pascale Cossart
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LPXTG protein InlJ, a newly identified internalin involved in Listeria monocytogenes virulence
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Role of lipid rafts in E-cadherin- and HGF-R/met-mediated entry of Listeria monocytogenes into host cells
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Inactivation of the srtA gene Listeria monocytogenes inhibits anchoring of surface proteins and affects virulence
Hélène Bierne
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Molecular Microbiology, 2002, 43 (4), pp.869-881
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InIB, a surface protein of Listeria monocytogenes that behaves as an invasin and a growth factor
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A role for cofilin and LIM kinase in Listeria-induced phagocytosis
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Journal of Cell Biology, 2001, 155 (1), pp.101-112
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The use of host cell machinery in the pathogenesis of Listeria monocytogenes
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Current Opinion in Immunology, 2001, 13, pp.96-103
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Impairment of lagging strand synthesis triggers the formation of a RuvABC substrate at replication forks
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EMBO Journal, 2001, 20 (3), pp.619-629
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The invasion protein InlB from Listeria monocytogenes activates PLC-gamma1 downstream from PI 3-kinase
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The invasion protein InIB from Listeria monocytogenes activates PLC-y1 downstream from PI 3-kinase
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Cellular Microbiology, 2000, 2 (6), pp.465-476
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Interaction between the protein InIB of Listeria monocytogenes and lipoteichoic acid: a novel mechanism of protein association at the surface of gram-positive bacteria
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Molecular Microbiology, 1999, 34 (5), pp.902-914
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The inlA gene of Listeria monocytogenes LO28 harbors a nonsense mutation resulting in release of internalin
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Infection and Immunity, 1998, 66 (7), pp.3420-3422
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InlB: An invasion protein of Listeria monocytogenes with a novel type of surface association
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uvrD mutations enhance tandem repeat deletion in the Escherichia coli chromosome via SOS induction of the RecF recombination pathway
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Molecular Microbiology, 1997, 26 (3), pp.557-567
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Deletions at stalled replication forks occur by two different pathways
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EMBO Journal, 1997, 16 (11), pp.3332-3340
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Isolation of a dnaE mutation which enhances RecA-independent homologous recombination in the Escherichia coli chromosome
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Competition between parental and recombinant plasmids affects the measure of recombination frequencies
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Flanking sequences affect replication arrest at the Escherichia coli terminator terB in vivo
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When replication forks stop
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Mechanisms of illegitimate recombination
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The replication termination signal terB of the Escherichia coli chromosome is a deletion hot spot
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