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A homoeostatic switch causing glycerol-3-phosphate and phosphoethanolamine accumulation triggers senescence by rewiring lipid metabolism
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Biological and Therapeutic Implications of the Tumor Microenvironment in Pituitary Adenomas
Mirela-Diana Ilie
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hal-04113463v1
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Loss of Pla2r1 decreases cellular senescence and age‐related alterations caused by aging and Western diets
Amélie Massemin
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hal-04225404v1
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Stiffness-induced cancer-associated fibroblasts are responsible for immunosuppression in a platelet-derived growth factor ligand-dependent manner
Pia Gamradt
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hal-04373776v1
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Pericyte stem cells induce Ly6G + cell accumulation and immunotherapy resistance in pancreatic cancer
Zhichong Wu
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hal-04047023v1
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Inhibition of activin signalling reduces the growth of LβT2 gonadotroph pituitary tumours in mouse
Audrey Ziverec
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scAN1.0: A reproducible and standardized pipeline for processing 10X single cell RNAseq data
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Exploring the MEN1 dependent modulation of caspase 8 and caspase 3 in human pancreatic and murine embryo fibroblast cells
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Lack of Tgfbr1 and Acvr1b synergistically stimulates myofibre hypertrophy and accelerates muscle regeneration
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Intratumoural spatial distribution of S100B + folliculostellate cells is associated with proliferation and expression of FSH and ERα in gonadotroph tumours
Mirela Diana Ilie
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EGFR-dependent aerotaxis is a common trait of breast tumour cells
Ivan Mikaelian
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hal-04045454v1
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PLA2R1 promotes DNA damage and inhibits spontaneous tumor formation during aging
Anda Huna
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inserm-03420490v1
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The scaffold protein menin is essential for activating the MYC locus and MYC‐mediated androgen receptor transcription in androgen receptor‐dependent prostate cancer cells
Yakun Luo
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Phospholipase A2 receptor 1 promotes lung cell senescence and emphysema in obstructive lung disease
Delphine Beaulieu
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inserm-03420591v1
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Reduced menin expression leads to decreased ERα expression and is correlated with the occurrence of human luminal B-like and ER-negative breast cancer subtypes
Romain Teinturier
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Stroma Involvement in Pancreatic Ductal Adenocarcinoma: An Overview Focusing on Extracellular Matrix Proteins
Sophie Liot
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hal-03281971v1
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Men1 disruption in Nkx3.1-deficient mice results in ARlow/CD44+ microinvasive carcinoma development with the dysregulated AR pathway
Romain Teinturier
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Myriam Decaussin-Petrucci
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Virginie Vlaeminck-Guillem
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hal-03666659v1
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Reinforcing one-carbon metabolism via folic acid/Folr1 promotes β-cell differentiation
Christos Karampelias
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hal-03666669v1
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Failed Apoptosis Enhances Melanoma Cancer Cell Aggressiveness
Kevin Berthenet
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hal-02999426v1
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Immune Landscape of Pituitary Tumors Reveals Association Between Macrophages and Gonadotroph Tumor Invasion
Moitza Principe
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Generation of a conditional transgenic mouse model expressing human Phospholipase A2 Receptor 1
Sara Jaber
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inserm-02865473v1
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Chromosomal instability in the prediction of pituitary neuroendocrine tumors prognosis
Hélène Lasolle
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Oncogene-Induced Senescence Limits the Progression of Pancreatic Neoplasia through Production of Activin A
Yajie Zhao
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hal-03008999v1
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Chromosomal instability in the prediction of pituitary neuroendocrine tumors prognosis
Hélène Lasolle
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Stromal protein βig-h3 reprogrammes tumour microenvironment in pancreatic cancer
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The Microenvironment of Pituitary Tumors—Biological and Therapeutic Implications
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Phospholipase D regulates the size of skeletal muscle cells through the activation of mTOR signaling.
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Assignment testing reveals multiple introduced source populations including potential ash hybrids (Fraxinus excelsior x F. angustifolia) in Ireland
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European Journal of Forest Research, 2013, 132, pp.195-209
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Reexpression of oncoprotein MafB in proliferative β-cells and Men1 insulinomas in mouse.
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Tif1γ Suppresses Murine Pancreatic Tumoral Transformation by a Smad4-Independent Pathway
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hal-02954464v1
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Reconstituted expression of menin in Men1-deficient mouse Leydig tumour cells induces cell cycle arrest and apoptosis
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European Journal of Cancer, 2007, 43 (2), pp.402-414
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Death by neglec as a deletional machanism of peripheral tolerance.
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Deletion of a C-terminal sequence of the class II-associated invariant chain abrogates invariant chains oligomer formation and class II antigen presentation.
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Invariant Chain Expression Similarly Controls Presentation of Endogenously Synthesized and Exogenous Antigens by MHC Class II Molecules
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Inhibition by chloroquine of the class II major histocompatibility complex-restricted presentation of endogenous antigens varies according to the cellular origin of the antigen-presenting cells, the nature of the T-cell epitope, and the responding T cell.
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